Josh Mollerus delivered the start at pitcher his coaching staff had been waiting for all summer, delivering seven strong innings and carrying a shutout through six of them as the Humboldt Crabs exposed the visiting California Expos in a 12-2 rout Friday night at the Arcata Ball Park.With the win the Crabs (11-10) are now above .500 for the first time this summer.After a few sub-optimal outings which saw Mollerus (1-2) struggle to make it through the first few innings the University of San …
Some recent findings might raise the eyebrows of perceptive readers: “How does that support evolution?”Marine reptile big bang: Science Daily describes another case of abrupt appearance in the fossil record. Marine reptiles like plesiosaurs and ichthyosaurs exploded onto the scene. First, we learn that “their origins and early rise to dominance have been somewhat mysterious.” Then we learn from paleobiologists at the University of Bristol that “they burst onto the scene, rather than expanding slowly into their ecosystems.” Yet, somehow, this is dubbed an “evolutionary burst.” Darwinians have known about this all along. Did they tell the public?Co-author Professor Michael Benton said: “We always knew that the marine reptiles expanded relatively fast into a world in turmoil, after a devastating mass extinction event that killed as many as 95 per cent of species. But what was unusual was that they were inventing entirely new modes of life that had not existed before the end-Permian mass extinction. Our work shows they expanded into nearly every mode of life, indicated by their feeding habits and range of body sizes, really much faster than might have been imagined.“If they “expanded” (i.e., evolved) much faster than might have been imagined, then by simple logic, it takes super-imagination to think they really did evolve that fast.Size doesn’t matter: If you make a male’s genitals bigger, do they outcompete the small guys? (Don’t get excited; we’re talking about mosquitofish here). Nature Communications tested the idea by artificially selecting fish with bigger, well, junk. It made no difference to their evolutionary fitness: there was “no effect of relative genital size on male reproductive success,” they say was their “main finding,” in contrast to previous claims. This was the case for both natural and sexual selection separately or in combination.Previous studies have reported directional selection on relative gonopodium length, but we did not find that males from up-selected lines are more attractive to females nor that they have weaker swimming performance than down-selected males. In combination, we did not find that the net effect of selection is greater male reproductive success (fitness) for control line males than males in either the up-selected or down-selected lines when they freely compete for mates and fertilization opportunities in semi-natural pools. In short, we did not find that novel genital–body size combinations are selected against.British microevolution: Claiming that “humans haven’t stopped evolving,” Nature reports on a study of “2,000 years of British evolution.” But do accentuations of traits like blue eyes, blond hair and ability to digest milk represent evolution? A Brit can still marry an African or Chinese person and have kids. The article calls the characteristic British traits “adaptations” without showing how mutation or selection increased their fitness. We’re all one species, and one race – the human race. Sorting out traits is not evolution in the sense Darwin meant.Differences are not necessarily evolved differences: 17 evolutionary anthropologists from multiple universities are apparently proud of their ability to measure differences between apes and humans. But is it evolution? In Nature, they paraded their measurements, showing that humans have higher metabolism, more body fat and longer childhoods than chimpanzees, gorillas and orangutans. That’s hardly news. On what basis, though, can they say this data represents “Metabolic acceleration and the evolution of human brain size and life history” without assuming what they think they are proving? Nature‘s editors committed the same fallacy in their Editorial: “humans evolve to use more energy.” That’s a teleological statement, forbidden by Darwinian theory, but Science Daily picked up the theme without any criticism whatsoever.Evolving proteins: Making Darwin proud, Science Daily announces, “Scientists discover the evolutionary link between protein structure and function.” OK, how, exactly? By just believing it. “The big problem in biology is the question of how a protein does what it does,” the lead author of a study says. “We think the answer rests in protein evolution.” They compared modern proteins with those preserved in soft tissue in fossils (that fact raising an important question about preservation for such long time periods). Did they find evolution? You be the judge:“It turns out that there are little snippets in our genes that are incredibly conserved over time,” Caetano-Anollés says. “And not just in human genomes. When we look at higher organisms, such as plants, fungi and animals, as well as bacteria, archaea, and viruses, the same snippets are always there. We see them over and over again.“Rather than explaining these by the neo-Darwinian processes of mutation and selection (terms they never use), they propose a model of “hierarchical modularity” as an “emergent property,” comparing it to the advance of artificial communication networks. That’s not Darwinian evolution; if anything, it’s intelligent design.Butterfly mimicry: The wing patterns on tropical butterflies are often used as examples of evolution. “Natural selection sculpts genetic information to limit diversity,” PhysOrg claims, but what was observed? In a word, epistasis – linked variations. The beneficial mutation causes simultaneous changes elsewhere in the genome that reduce further evolution. This presents a puzzle for Darwinian theory. If a change reduces diversity, how is the butterfly going to evolve further? The researchers overcame this challenge by imagining what could happen in the future. It’s intriguing. The Darwinians used intrigue to keep their theory alive.Intriguingly, the study effectively suggests that natural selection could limit a species’ ability to adapt to future environmental change by removing linked variations that, despite having no immediate beneficial impact on the species, could become relevant to its survival and capacity to cope with its environment in the future.“Variation is a kind of raw material and you don’t necessarily use it all at any one time,” Martin explained. “It’s something that could be used to adapt and change in the future.“For more about epistasis and selection, see a PNAS paper, “The effect of gene interactions on the long-term response to selection.” It begins, “The role of gene interactions in the response to selection has long been a controversial subject” – a surprising statement since that was supposedly solved in the 1930s. The paper shows the dominance of epistasis and genetic drift, factors that confuse simplistic ideas about progress by mutation and natural selection.Still trying to figure Darwin out: What do flying birds have to do with bacteria? Not much, most might answer. Science Daily, however, titles an article, “How did birds get their wings? Bacteria may provide a clue, say scientists.” Taking the bait, we look inside the package and find that biologists are still trying to figure out the basic ideas of evolution that we were all taught Darwin and his disciples solved.The evolution of major novel traits — characteristics such as wings, flowers, horns or limbs — has long been known to play a key role in allowing organisms to exploit new opportunities in their surroundings.What’s still up for debate, though, is how these important augmentations come about from a genetic point of view.Instead of studying how birds evolved Flight, though, Oxford scientists used divination tools on bacterial genes. “The researchers allowed 380 populations of Pseudomonas aeruginosa bacteria to evolve novel metabolic traits such as the ability to degrade new sugars,” the article says. “This gave the researchers the opportunity to witness evolution happening in real-time.” Then we find out that the genes to digest these sugars were already there or were acquired by horizontal gene transfer. The birds, meanwhile, were left waiting on the ground for their lucky mutation.LGBT beetles: Science Daily presumes to tell readers “Why animals court their own sex,” admitting this is a Darwinian conundrum: “Same-sex sexual behavior is common in animals but puzzles evolutionary biologists since it doesn’t carry the same obvious benefits as heterosexual courtship behavior that leads to mating and production of offspring.” Proposing to give a new Darwinian explanation, the article promises that “A study sheds new light on the pervasiveness of same-sex sexual behavior in the animal kingdom.” The issues of whether SSB [same sex behavior] in animals has anything to do with human homosexual behavior will be shelved for the time being.The light-shedders at Uppsala University propose this idea: “The researchers hypothesized that, because males and females share most of their genes, SSB [same-sex behavior] may occur in one sex because its underlying genes carry benefits when expressed in the other.” Key word: may occur. “The findings thus support the idea that SSB may be prevalent in one sex because the genes regulating the behaviour are preserved by natural selection through their benefits in the opposite sex, pointing to a general mechanism maintaining multiple forms of SSB across a wide variety of animals.” Did they study this in a wide variety of animals? No; just in beetles, where they artificially selected the ones that seemed to show SSB. Their siblings seemed to have more offspring. But that could be due to a simple increase in overall mounting behavior by all the beetles, not because natural selection was somehow preserving SSB. If they think they shed light on evolution, they only did so by shedding the light into the trash can.How birds got their red color: This tale made the rounds this week. Cardinals and other birds with bright red feathers didn’t really evolve the ability to make red pigment. A study described by Science Daily finds that “nearly all birds have the latent capacity to make red feathers, but in order to actually do so, they must evolve the means of expressing [this gene] in the skin in addition to the retina.” True to the Stuff Happens Law, “dazzling red colors have evolved repeatedly in this group, mostly by the mechanism described here, but there are some very interesting exceptions.” Another Science Daily piece says that two color morphs can appear on the same bird “despite the pressures of evolution.” Amazing but true; “This is an important finding and helps evolutionary biologists understand how multiple color varieties can co-exist together in the face of natural selection.” Source paper: Current Biology.Non-evolution as evidence for evolution: Another case of gene conservation was noted this week in Science Daily: an enzyme necessary for construction of mitochondria has been “conserved throughout evolution“. No one seems to notice the statement is self-contradictory, as if to claim a pilot was grounded throughout his flight career. He never had a flight career if the statement is true.Now it’s your turn. You’ve watched some Baloney Detecting on the above evolutionary claims; now try your hand at these others. Teachers may use this as a class assignment for middle or high-school grade levels. Demonstrate a couple in class, then assign some others for homework. The evolution of sauropod dinosaurs (PhysOrg) [G]ene provides evidence that our limbs may have evolved from sharks’ gills (PhysOrg) Plants are ‘biting back’: Sting of the rock nettle (Science Daily) New evidence connects dung beetle evolution to dinosaurs (Science Daily) Tips: Watch for the phrase “evolved to” – this is verboten, because it is teleological. Darwinian evolution has no goal, no plan, and no purpose. Watch for instances where the empirical evidence is counter to what was expected. Look for evidence of abrupt appearance and conservation. Are the authors inventing meaningless phrases that assume evolution, like “metabolic acceleration” or “evolutionary stasis”? Discard all references that assume evolution occurred; what is the real evidence? Can you rescue something scientifically useful after the data are rinsed of Darwin-colored whitewash?Darwinian evolution is the scam of our age. All data are forced to support evolution, because alternatives have been ruled out of bounds. Natural selection is a fancy phrase for the Stuff Happens Law. And lazy Darwinians feel they have done their job by just saying, “It evolved.” We need to raise up an army of skilled Baloney Detectors who are not fooled by bluffing and evasion. Join the revolution against evolution; help us overturn the dork side of the farce manipulated by its naked Emperor Charlie. Survival of the oldest: Why do some species make it when some don’t? (Science Daily) The evolution of development (PhysOrg) Role of life’s timekeeper—a novel theory of animal evolution (PhysOrg) How plants conquered the land (Science Daily) How and why single cell organisms evolved into multicellular life: The story of the evolution of life (Science Daily) (Visited 51 times, 1 visits today)FacebookTwitterPinterestSave分享0
Mandela day is about coming together, taking action, and in the process bringing to life Nelson Mandela’s legacy (Image: Habitat for Humanity)67 houses in one week – this is the target that’s been set for International Nelson Mandela Build Week, an ambitious project that will run from 14 to 18 July in Pelican Park, the largest integrated development project in the Western Cape.The drive will include a 24-hour build on the fourth day of the week in the lead-up to Nelson Mandela International Day on Friday, 18 July. Thursday, 17 July will see teams of volunteers working around the clock in a 24-hour “blitz build” to exceed the target of 67 new houses in one week.The project, run by Habitat for Humanity South Africa in partnership with the Nelson Mandela Foundation, will honour the Mandela Day call for individuals and organisations to take action to change the world for the better and, in doing so, to help build a global movement for good.“To achieve this, Habitat needs your help and is asking for your support in helping to raise the required funding and also volunteering your time on-site to build the houses,” says Habitat marketing manager Adrienne Burke.“Working together under the guidance of qualified artisans, volunteers, beneficiaries and community members will lend a hand in mixing cement, passing building blocks, erecting, plastering and bagging walls, to name just a few of the tasks at hand.”Mandela Day, the final day of the build week, will be commemorated with the official handover of the 67 houses to their new owners.Burke said the day would “signify South Africans coming together, taking action, and in the process bringing to life Nelson Mandela’s legacy in his values, his vision and his leadership, moving several steps closer to building the country of our dreams.”Individuals or organisations wishing to be part of International Nelson Mandela Build Week can contact Adrienne Burke at [email protected] or e-mail [email protected]
29 July 2014The United States is to set up centres to train Africa’s next generation of political and economic leaders – starting next year with centres in Senegal, Ghana, South Africa and Kenya – as it moves to expand US President Barack Obama’s Young African Leaders Initiative.Obama made this announcement in Washington on Monday during a meeting with a group of 500 young Africans who were chosen from nearly 50 000 applicants from across the continent to participate in the Washington Fellowship exchange programme, part of the Young African Leaders Initiative.He also announced that the exchange programme – which gives Africans aged 25 to 35 the opportunity to study at the top 20 universities in the US – was being renamed the Mandela Washington Fellowship for Young African Leaders, in honour of the former South African president.According to news agency Bloomberg, the US Agency for International Development would be providing $38-million to set up the first four leadership centres. Obama said that US universities, African institutions and business partners like Microsoft and MasterCard Foundation would also be partnering on setting up and running the centres.“Starting next year, young Africans can come to these centres to network, access the latest technology, and get training in management and entrepreneurship,” Obama said. “The first centers will be located in Senegal, Ghana, South Africa and Kenya – and will provide tens of thousands of young Africans the resources they need to put their ideas into action.”According to the White House, the Young African Leaders Initiative was launched by Obama in 2010 “as part of a long-term investment in the next generation of African leaders. It aims to sharpen their skills, to improve their networks, and to strengthen partnerships between the United States and Africa for years to come”.Obama told his audience on Monday that “even as we acknowledge the real hardships that so many Africans face every day, we have to make sure that we’re all seizing the extraordinary potential of today’s Africa, the youngest and fastest-growing continent.”The Young African Leaders Initiative, he said, “is about capitalizing on the creativity and talent of Africa’s young leaders by empowering them with the skills, training, and technology necessary to make lasting change and meaningful progress back home.”Next week, Obamba will be hosting the first US-Africa Leaders Summit, representing the largest gathering that any American president has hosted with African heads of state and government.SAinfo reporter
Share Facebook Twitter Google + LinkedIn Pinterest Malignant Lymphosarcoma is the most common neoplastic (cancerous) disease identified in cattle slaughtered in the United States and largest single reason cattle are condemned during postmortem inspection. A 2009 report sites malignant lymphosarcoma for 13.5% of beef cattle condemnations and 26.9% of dairy carcass condemnations. The bovine leukemia virus (BLV) initiates the cancer and this virus routinely spreads through contact with blood from an infected animal. BLV can spread through procedures such as injections with used needles, surgical castration and/or dehorning, tattooing, rectal palpation with dirty sleeves, as well as through insect vectors such as horseflies. Calves may also be exposed during pregnancy or while nursing an infected dam.Less than 2% of BLV-infected animals will go on to develop lymphosarcoma, a cancer affecting lymph nodes, multiple organs and white blood cells. Tumors may occur in the spinal canal, uterus, heart, abomasum, kidney and/or lymph nodes. The most common clinical signs include anorexia, weight loss and fever. Bovine leukosis is not transmissible to humans.Although cancer in cattle is not treatable, the risk can be substantially decreased with proper management procedures. Bovine leukemia virus (BLV) is a common viral infection in US cattle, with 39% of beef cow-calf herds and 83% of dairy herds containing BLV-infected cattle. In contrast, many European countries have successfully eradicated BLV from their herds. BLV-infected animals become permanent carriers. This retrovirus causes the cattle disease known to veterinarians as “Enzootic Bovine Leukosis” or EBL. EBL has three disease stages: asymptomatic, persistent lymphocytosis (PL), and finally the cancerous stage (leukemia or lymphoma). Cattle and sheep are thought to be the only species naturally susceptible, and prevalence rates are higher in dairy breeds than beef. The majority of infected animals are over two years old, with younger animals developing “sporadic bovine leucosis.” Sporadic forms (juvenile, thymic and cutaneous) are unrelated to the bovine leukemia virus.Persistent lymphocytosis is defined by an increase in total lymphocyte count three times above normal, and persisting for at least three months but with no clinical signs of neoplastic (cancerous) lesions. PL is thought to occur in approximately a third of all BLV-infected cattle. Recent research suggests BLV infection causes abnormal immune function however little is known regarding its specific effect on immunity and resistance to other infectious diseases. It definitely impairs milk production and cow longevity as infected cattle are often culled or die at an early age.The final stage of Enzootic Bovine Leukosis is cancer, usually malignant lymphosarcoma. Only a small fraction of BLV-infected cattle, approximately 1% to 2%, develop lymphosarcoma after an average incubation period of seven years after infection with the virus. The sites most commonly affected by tumors are the heart, abomasum (true stomach), uterus, kidney, spinal cord, and the area behind the eyeball. Early signs include loss of appetite, weight loss, decreased milk production, fever, and enlarged lymph nodes. Some infected cattle will show no sign of the disease as approximately 5% to 20% of cases present as sudden death. Other clinical signs of disease are linked to the organ system affected by tumor metastasis and can include:Tumors in many parts of the body, which can appear as lumps under the skin. The most common presentation is that of enlarged lymph nodes which look like fist-size swellings in front of the shoulder, in the flank and other locations. This is often the earliest indicator of disease.Tumors in the heart can cause cardiovascular dysrhythmmias, jugular vein distension, rapid heart rate or murmurs.Abomasal (stomach) tumors cause gastrointestinal problems digesting feed resulting in loss of appetite and weight, constipation or diarrhea, abomasal tympany or bloat, and hypomotility.Retrobulbar tumors can cause bulging eyes, blindness and other ocular signs.Spinal tumors may cause rear limb weakness or paralysis. Neurological signs and lameness also sometimes occur due to local tumor growth.How is BLV spread? Transmission of bovine leukemia virus is mainly horizontal from cow to cow. Routine cattle working procedures are an important route as they transfer contaminated blood between individuals on surgical equipment, needles and gloves. Rectal palpation without changing sleeves, the re-use of a single needle on multiple cows, dehorning, tattooing, and ear tagging without proper sanitation between animals are all are implicated in virus transmission. Biting flies and other blood-feeding insects are also proven modes of transmission. Direct transmission between animals in close contact occurs when virus-infected blood, fluids or tissues enter through small cuts in the skin. This includes using bulls for breeding because there is occasionally blood exchange due to minor penile or vaginal trauma. Vertical transmission from cows to baby calves during pregnancy or when nursing is less common but the virus can pass via the placenta, during delivery and in colostrum.How is BLV diagnosed? Blood testing is the first step to identify BLV-positive (infected) animals. An inexpensive ($4) serum ELISA test is available at the UK Veterinary Diagnostic Laboratory to detect antibodies to the virus. Once an animal is infected and tests positive, she will remain test positive for her lifetime. Testing can be done in animals over six months of age. Lymphosarcoma (the actual cancer) is best diagnosed through a lymph node biopsy.What should a producer do with a BLV + animal? The decision on what to do with a positive animal is best determined through consultation with the herd veterinarian, based on the specific goals of the individual farm. Implementation of a BLV control program depends largely on a comparison of the cost of the disease and the cost of preventing disease. Economic losses stem from the inability to sell cattle for export or as bull studs, condemnation of carcass at slaughter if tumors are present, and clinical disease/death loss.Recent research has shown lost milk production realized in lower weaning weights, higher culling rates, and immune system dysfunction also need to be considered. Seed stock producers and especially those who export internationally are more likely to aggressively pursue BLV-free herd status. Disease control is based on strict biosecurity that eliminates contact with infected blood, the most important measure for preventing the spread of BLV in cattle herds. Segregating or culling positive cattle, adding only BLV-negative cattle to the herd, making management changes to reduce transmission such as single-use needles/sleeves and disinfecting equipment between animals, and implementing an integrated pest management program will substantially reduce the risk of this and all other blood-borne diseases.
Share Facebook Twitter Google + LinkedIn Pinterest Check back daily for photos and results from the 2017 Ohio State Fair Draft Horse show.CLICK HERE FOR THE GOVERNOR’S CUP VIDEOMonday, July 31, 2017Afternoon Show Photos Evening Show Photos Official Results from Monday’s Show ALL MONDAY RESULTS IN PDF FORMATTuesday, Aug. 1, 2017 Official Results from Tuesday’s Show ALL TUESDAY RESULTS IN PDF FORMATWednesday, Aug. 2, 2017 Official Results from Wednesday’s Show ALL WEDNESDAY RESULTS IN PDF FORMATThursday, Aug. 3, 2017 Official Results from Thursday and Friday Shows THURSDAY AND FRIDAY MORNING AND AFTERNOON RESULTS IN PDF FORMATOfficial Results from Friday Evening FRIDAY EVENING RESULTS IN PDF FORMAT
Import and Interpret Your Footage in PremiereSelect the high-frame-rate footage you’d like to encode, and right-click.Select Modify > Interpret FootageIn the dialog box, under the Frame Rate tab, select the radio button “Assume this frame rate,” and input your desired frame rate.Click OK.Create Your Proxies Using Media EncoderNow it’s time to transcode your high-frame-rate footage into proxies using Adobe Media Encoder.Select the conformed clips in your Project Panel, and right-click.Navigate to Proxy > Create Proxies. Select your desired Proxy configuration. The Quicktime preset 1280×720 GoPro CineForm works well for most UHD projects.Select OK.At this point, Adobe Media Encoder will open and your clips will begin to populate the Render Queue.Once your clips load into Media Encoder, stop the transcode by pressing the red square in the top-right corner of the window. Do not finish the current job. Be sure to reset the status of the unfinished transcode by right-clicking the clip and selecting “Reset Status.”Next, select all of the high-frame-rate footage in the Render Queue and right-click.Select “Interpret Footage.” In the dialog box, under the Frame Rate tab, select the radio button “Assume this frame rate,” and input your desired frame rate.Select OK.Hit the green play button in the top-right corner of the window to begin the transcode.Check proxy status, and test your clipsAfter the transcode is complete, navigate to the Project Panel in Premiere. The proxy status for your transcoded clips should have changed. You can toggle the Proxy Status display in Premiere’s Project Panel by right-clicking the column headers and selecting “Metadata Display” and adding the “Proxy” display from the list.You’ll have to attach the completed proxy to your first clip (that was cancelled at the beginning of the transcode). You can do this by right-clicking this clip and navigating to Proxy > Attach Proxies and selecting the proxy created in the transcode.Interested in the tracks we used to make this video?“In the Light in the Dark” by Ferry Island Music“A Vibe Called Quest” by Sounds Supreme“Technology of the Future” by Denis“Street Signs” by Sound ForceLooking for more post-production tips and tricks? Check out these articles.The Audio Ramp-Up: The Best Transition You Should Be UsingThe 3 Easiest Ways to Cut Clips in Adobe Premiere Pro CCRoundup: 5 Awesome Editing Effects in Adobe Premiere ProIsolating Image Channels to Work with Chroma and Luma in PremierePost-Production Tip: Quick and Dirty Noise Reduction Without a Plugin Working with proxy files in Adobe Premiere Pro is easy, but high frame rate footage requires a few extra steps. Here’s what you need to know.With droves of mirrorless cameras like the GH5 and Fuji X-T3 hitting the market at affordable prices, filmmakers and videographers can shoot 4K at 60fps, and beyond. For these shooters, proxy workflows are a mainstay in post-production.Adobe Premiere makes it easy to create, attach, and edit with proxy files. For example, when working with non-high-frame-rate footage, the proxy workflow is relatively straightforward:Import your footage into Premiere.Interpret your footage (or use Premiere’s proxy ingest settings).However, when working with high-frame-rate footage you’ll need to follow some additional steps.Use this as a guide for organizing, ingesting, and creating high-frame-rate proxies for editing in Premiere.
Giving a tough competetion to BMW X1 and Audi Q3, Mercedes Benz is all set to steal hearts in India.Mercedes Benz B Class Sports Tourer will hit Indian roads on Wednesday. The car was apparently seen at Buddh International Circuit. The Mercedes Benz has some quality features:- Looks amashing with an classy dashboard- AMG style steering wheel- Can accelerate from 0 to 100 kmph in 10.2 secondsMercedes Benz will be priced at sub Rs.25 lakh level as per reports.
zoomIllustration. Image Courtesy: Pixabay under CC0 Creative Commons license Monaco-based Navios Maritime Containers took delivery of the Navios Miami, a 2009-built containership, on September 12, 2018.As World Maritime News previously reported, the 4,563 TEU vessel was acquired for a price of USD 13.9 million.As informed, the vessel is chartered out at a net rate of USD 11,791 per day until December 2018.Navios Miami is expected to generate approximately USD 1.9 million of EBITDA for the first year.The acquisition of the vessel was financed with cash on the balance sheet and a USD 9 million term loan facility. The debt has an amortization profile of seven years, matures in September 2023 and bears interest at LIBOR plus 300 bps per annum, according to the company.Earlier this month, the company announced plans to pursue a direct listing on the US Stock Exchange after it ceased marketing its proposed US initial public offering.Navios Containers controls 26 vessels, totaling 124,101 TEU, with a current average fleet age of 10.4 years.
On Sunday 21st February 2016, Jude Law, Juliet Stevenson, Tom Odell, Sir Tom Stoppard, Matt Berry, Shappi Khorsandi and Toby Jones alongside a number of displaced refugees, took to the stage to perform a one off Letters Live performance in association with Good Chance at the refugee camp in Calais, France.Barbak (a refugee), Jude Law, Shappi Khorsandi and Toby Jones at the Good Chance theatre in the Calais CampCredit/Copyright: Help RefugeesThe performance was held in aid of Help Refugees, a humanitarian organisation providing much of the aid on the ground, and took place at Good Chance Theatre, a temporary theatre based in the Jungle which offers a varied programme of art, theatre and music events.Ten letters from the renowned Letters Live series were staged alongside new writing penned and performed by refugees currently living in the jungle, read in English and translated into Arabic, Farsi, Pashtu and Kurdish. There was live music from the the camp’s most popular rappers and musicians, as well as Tom Odell, Maria Friedman and Adrian der Gregorian who rounded off the event.The show included a reading of a letter to David Cameron which was delivered earlier this week as well as Ghandi’s letter to Hitler ‘For the sake of humanity’ and ‘We Are Just Ordinary People’ a letter from a refugee to the people of Europe.Juliet Stevenson read her heartfelt message to the refugees saying: “You have already made incredible journeys to get here, journeys that required strength and courage that we can only dream of possessing. I am so ashamed of the Europe that has refused you and of the politicians who have failed you. They do not speak for us. There is another Europe, which will fight on your behalf, and where you will always be welcome.”Liz Clegg, a British volunteer who runs the makeshift Women and Children’s Centre in the camp read a letter to one of the young unaccompanied boys she cares for, telling him of her fears for him, adding “I can’t promise to solve this, but I am trying…I will fight to the bitter end to keep you safe”.Performed to an audience of around 200 refugees the unique and unprecedented event was mounted two days before the proposed official eviction of more than 3500 people from the southern section of the Calais camp, which is being challenged by Help Refugees, L’Auberge des Migrants and several other organisations on the ground in the French courts, with a hearing set for Tuesday in Lille at 2pm.The section of the camp which is due to be demolished is largely occupied by unaccompanied children and families. A recent census conducted by L’Auberge des Migrants & Help Refugees found over 3,000 people and more than 440 children living in this section of the camp, 291 of which are unaccompanied. Over 100 of the unaccompanied children are currently being helped by Citizens UK to claim their legal right to be reunited with their families in the UK.Letters Live, Good Chance, Help Refugees and Citizens UK hope that today’s performance will raise awareness of the plight facing these refugees.Speaking of their experience,Jude Law said: “It’s our responsibility as humans to look after our children. The children in the camp at Calais need us. It isn’t a big ask. It is simply the right thing to do.”Taj, a Afghani refugee from the camp who performed at the event said: “We are not soldiers, we are not politicians, we are not terrorists. We are ordinary people who want to live a peaceful life. Please open your hearts and borders to us, and help to end the war.”Sir Tom Stoppard and Sabrina Stoppard said: “An inspiring and humbling day, a wonderful day and we regret to say, shaming too.”Tom Odell said: “Today was one of the most humbling and inspiring experiences of my life. Something needs to be done.”Help Refugees said: “We are overwhelmed by the level of support from everyone coming together today in a show of solidarity to highlight the plight facing the refugees in Calais and Dunkirk. The bulldozing of the camp must be postponed until the children have adequate and safe alternatives, which are currently not on offer. We would like to thank Letters Live, all the British artists who performed, Good Chance, and most of all the refugees who took part in today.”Joe Murphy and Joe Robertson of the Good Chance Theatre said: “Today was an example of the energy that can be produced if we work together. This is an unjust set of circumstances. Children should not live like this in any country of the world. We want this day to draw deserved attention to the situation these people find themselves in. It was a pleasure to work with Help Refugees, Citizens UK and Letters Live to achieve this moment.”Earlier this week Letters Live, Help Refugees, Citizens UK and Good Chance delivered a letter to David Cameron calling for urgent action to delay the demolition of the southern part of the camp to allow time for intervention to ensure adequate protection of the camps refugee children. More than 110,000 people including over 150 public figures have now signed this letter.Find out more here.Source:Help Refugees